Proteomics in Computational Systems Biology

A large number of genes with diverse normal functions are involved in human cancers. More than 500 genes have been identified as strongly implicated in the process of transforming normal cells to cancer cells. The expression of these genes in normal cells contributes to normal growth, survival and function, whereas dysregulated expression, including overexpression, loss of expression or expression of a defect protein, in cancer cells contributes to ungoverned tumor growth. Altered gene expression can be caused by coarse structural and numerical chromosomal rearrangements, specific gene amplifications, silencing of transcription through methylation and mutations.

Relevant Conferences:

14th World Congress on Cancer Therapy December 05-07, 2016 Philadelphia, Pennsylvania, USA; 7th International Conference on Proteomics October 24-26, 2016 Rome, Italy; 2nd International Conference on Genetic and Protein Engineering November 14-16, 2016 Atlanta, Georgia, USA; International Conference on Amino Acids and Proteins December 08-09, 2016 Dallas, Texas, USA; 7th International Conference on Metabolomics November 14-16, 2016 Orlando, Florida, USA; 8th International Conference on Proteomics March 20-22, 2017 Philadelphia, Pennsylvania, USA; XI National Conference of the Italian Proteomic Association May 16-19, 2016 Perugia, Italy; HUPO 15th Annual World Congress September 18-22, 2016 Taipei, Taiwan; X Annual Congress of the European Proteomics Association June 21-25, 2016 Istanbul, Turkey; US HUPO 12th Annual Conference March 13-16, 2016 Boston, Massachusetts; HUPO 16th Annual World Congress September 17-20, 2017 Dublin, Ireland.

  • In silico systems biology
  • Biological computations
  • Network & synthetic Biology
  • Systems biologists & biomedicine
  • Cancer systems biology and pharmacology
  • Developmental biology
  • Mathematical and computational biology
  • Systems biology: Global regulation of gene expression
  • Systems biology in drug development

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